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Ultra Peel Forte is for those with resilient skin who have more dramatic wrinkling, sun damage and residual scarring from breakouts are the most suitable candidates for this deeper and more active treatment. Ultra Peel® Forte will effectively treat aging skin, breakouts, pigment disorders, smoker’s skin and photo-damaged skin. This treatment is ideal for skin in need of rejuvenation. Excellent for clients who have not professionally exfoliated in over a year!
Trichloroacetic Acid (TCA) (20%) – is a peeling agent commonly used to remove fine lines, wrinkles and acne scarring.
Lactic Acid (10%) – is an alpha hydroxy acid naturally found in milk and sugars. It breaks down bonds between cells (desmosomes) to allow for easier exfoliation of dead surface cells while hydrating the skin. It also functions as a melanogenesis inhibitor by suppressing the formation of tyrosinase.
Kojic Acid – is an antibacterial agent and melanogenesis inhibitor that is naturally found in mushrooms. Its ability to chelate copper away from tyrosinase and decrease the number of melanosomes and dendrites makes it highly effective in reducing hyperpigmentation.
Azelaic Acid – is an effective melanogenesis inhibitor that helps to brighten uneven complexions. Azelaic acid is also effective for fighting breakouts because of its bactericidal and antimicrobial action. It has exfoliating and disinfecting properties that are most effective when used in combination with alpha hydroxy acids. Azelaic acid helps to normalize keratinization in the skin.
L-Ascorbic Acid (Vitamin C) – is a powerful water-soluble antioxidant that is the only bio-available form of vitamin C for the skin. It is also a cofactor for an enzyme crucial in the synthesis of collagen (prolyl hydrolase) and is an antioxidant agent that provides important protection against damage induced by UV radiation. Vitamin C also improves skin elasticity and decreases wrinkles by stimulating collagen synthesis. Additionally, it reduces erythema and acts as a melanogenesis inhibitor. It functions as an MMPi by controlling the expression of inflammatory enzymes